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<meta name="DC.Description" content="Summary: We herein report the case of a 53-year-old woman with Behcet’s disease and an acute T2-hyperintense lesion in left side of the pons. Echo-planar “trace” diffusion MR imaging revealed high signal intensity changes at the lesion site on b = 1000 s/mm2 images, initially suggesting restricted diffusion. On corresponding apparent diffusion coefficient maps, however, the lesion had high signal intensity and high apparent diffusion coefficient values (1.22 × 10−3 mm2/s), compared with the contralateral normal side of the pons (0.86 × 10−3 mm2/s) and compared with the normal temporal white matter (0.80 × 10−3 mm2/s). This was consistent with the presence of increased diffusion, hence vasogenic edema. Proton MR spectroscopy excluded acute infarction. This particular pattern (high signal intensity on b = 1000 s/mm2 images in association with high apparent diffusion coefficient values) likely represented the acute inflammatory process associated with disrupted brain-blood barrier in the fulminant form of neuro-Behcet’s disease. Follow-up examinations ≤2 years later revealed a remaining gliotic lesion." />
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<meta name="twitter:description" content="Summary: We herein report the case of a 53-year-old woman with Behcet’s disease and an acute T2-hyperintense lesion in left side of the pons. Echo-planar “trace” diffusion MR imaging revealed high signal intensity changes at the lesion site on b = 1000 s/mm2 images, initially suggesting restricted diffusion. On corresponding apparent diffusion coefficient maps, however, the lesion had high signal intensity and high apparent diffusion coefficient values (1.22 × 10−3 mm2/s), compared with the contralateral normal side of the pons (0.86 × 10−3 mm2/s) and compared with the normal temporal white matter (0.80 × 10−3 mm2/s). This was consistent with the presence of increased diffusion, hence vasogenic edema. Proton MR spectroscopy excluded acute infarction. This particular pattern (high signal intensity on b = 1000 s/mm2 images in association with high apparent diffusion coefficient values) likely represented the acute inflammatory process associated with disrupted brain-blood barrier in the fulminant form of neuro-Behcet’s disease. Follow-up examinations ≤2 years later revealed a remaining gliotic lesion." />
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      <h1 class="highwire-cite-title" id="page-title">Neuro-Behcet’s Disease: Diffusion MR Imaging and Proton MR Spectroscopy</h1>  
    	<div class="highwire-cite-authors"><span  class="highwire-citation-authors"><span class="highwire-citation-author first" data-delta="0">R. Nuri Sener</span></span></div>
  
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<div id="hw-article-author-popups-top-node-10983--61944428324" style="display: none;"><div class="author-tooltip-0"><div class="author-tooltip-name">R. Nuri Sener </div><ul class="author-tooltip-find-more"><li class="author-tooltip-gs-link first"><a href="/lookup/google-scholar?link_type=googlescholar&amp;gs_type=author&amp;author%5B0%5D=R.%2BNuri%2BSener%2B" target="_blank" class="" data-icon-position="" data-hide-link-title="0">Find this author on Google Scholar</a></li><li class="author-tooltip-pubmed-link"><a href="/lookup/external-ref?access_num=Sener%20RN&amp;link_type=AUTHORSEARCH" target="_blank" class="" data-icon-position="" data-hide-link-title="0">Find this author on PubMed</a></li><li class="author-site-search-link last"><a href="/search/author1%3AR.%2BNuri%2BSener%2B" rel="nofollow" class="" data-icon-position="" data-hide-link-title="0">Search for this author on this site</a></li></ul></div></div></div>  </div>

  
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        	<div class="highwire-cite-metadata"><span  class="highwire-cite-metadata-volume highwire-cite-metadata">Vol. 24</span>, <span  class="highwire-cite-metadata-issue highwire-cite-metadata">Issue 8 </span><div  class="highwire-cite-metadata-pubdate highwire-cite-metadata">1 Sep 2003 </div></div>
      
      
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