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<meta name="DC.Title" content="Unilateral Calcification of the Caudate and Putamen: Association with Underlying Developmental Venous Anomaly" />
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<meta name="DC.Description" content="SUMMARY: Stenosis of a DVA may result in chronic venous ischemia. We present 6 patients (3 men, 3 women; age range, 30–79 years; mean age, 53 years) with unilateral calcification of the caudate and putamen on noncontrast CT. This calcification typically spared the anterior limb of the internal capsule. No patient presented with symptoms referable to the basal ganglia or had an underlying metabolic disorder or other process associated with calcium deposition. All patients subsequently underwent gadolinium-enhanced MR imaging and/or CTA or conventional angiography demonstrating the presence of an adjacent DVA. We hypothesize that chronic venous ischemia in the drainage territory of the DVA causes the abnormal mineralization. Greater recognition of this entity will prevent misinterpretation of this finding as acute hemorrhage and will prevent unnecessary and sometimes invasive evaluation in such patients. Furthermore, this entity should be considered in the differential diagnosis of unilateral basal ganglia hyperattenuation.

AVM
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C
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Ca2+
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CTA
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HA
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ICH
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&lt;p&gt;Stenosis of a DVA may result in chronic venous ischemia. We present 6 patients (3 men, 3 women; age range, 30–79 years; mean age, 53 years) with unilateral calcification of the caudate and putamen on noncontrast CT. This calcification typically spared the anterior limb of the internal capsule. No patient presented with symptoms referable to the basal ganglia or had an underlying metabolic disorder or other process associated with calcium deposition. All patients subsequently underwent gadolinium-enhanced MR imaging and/or CTA or conventional angiography demonstrating the presence of an adjacent DVA. We hypothesize that chronic venous ischemia in the drainage territory of the DVA causes the abnormal mineralization. Greater recognition of this entity will prevent misinterpretation of this finding as acute hemorrhage and will prevent unnecessary and sometimes invasive evaluation in such patients. Furthermore, this entity should be considered in the differential diagnosis of unilateral basal ganglia hyperattenuation.&lt;/p&gt;" />
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<meta name="twitter:title" content="Unilateral Calcification of the Caudate and Putamen: Association with Underlying Developmental Venous Anomaly" />
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<meta name="twitter:description" content="SUMMARY: Stenosis of a DVA may result in chronic venous ischemia. We present 6 patients (3 men, 3 women; age range, 30–79 years; mean age, 53 years) with unilateral calcification of the caudate and putamen on noncontrast CT. This calcification typically spared the anterior limb of the internal capsule. No patient presented with symptoms referable to the basal ganglia or had an underlying metabolic disorder or other process associated with calcium deposition. All patients subsequently underwent gadolinium-enhanced MR imaging and/or CTA or conventional angiography demonstrating the presence of an adjacent DVA. We hypothesize that chronic venous ischemia in the drainage territory of the DVA causes the abnormal mineralization. Greater recognition of this entity will prevent misinterpretation of this finding as acute hemorrhage and will prevent unnecessary and sometimes invasive evaluation in such patients. Furthermore, this entity should be considered in the differential diagnosis of unilateral basal ganglia hyperattenuation.

AVM
:   arteriovenous malformation
C
:   caudate
Ca2+
:   calcification
CTA
:   CT angiography
DVA
:   developmental venous anomaly
HA
:   headache
ICH
:   intracerebral hemorrhage
L
:   left
NA
:   not applicable
P
:   putamen
R
:   right
SZ
:   seizure" />
<meta name="og-title" property="og:title" content="Unilateral Calcification of the Caudate and Putamen: Association with Underlying Developmental Venous Anomaly" />
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<meta name="og-description" property="og:description" content="SUMMARY: Stenosis of a DVA may result in chronic venous ischemia. We present 6 patients (3 men, 3 women; age range, 30–79 years; mean age, 53 years) with unilateral calcification of the caudate and putamen on noncontrast CT. This calcification typically spared the anterior limb of the internal capsule. No patient presented with symptoms referable to the basal ganglia or had an underlying metabolic disorder or other process associated with calcium deposition. All patients subsequently underwent gadolinium-enhanced MR imaging and/or CTA or conventional angiography demonstrating the presence of an adjacent DVA. We hypothesize that chronic venous ischemia in the drainage territory of the DVA causes the abnormal mineralization. Greater recognition of this entity will prevent misinterpretation of this finding as acute hemorrhage and will prevent unnecessary and sometimes invasive evaluation in such patients. Furthermore, this entity should be considered in the differential diagnosis of unilateral basal ganglia hyperattenuation.

AVM
:   arteriovenous malformation
C
:   caudate
Ca2+
:   calcification
CTA
:   CT angiography
DVA
:   developmental venous anomaly
HA
:   headache
ICH
:   intracerebral hemorrhage
L
:   left
NA
:   not applicable
P
:   putamen
R
:   right
SZ
:   seizure" />
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      <h1 class="highwire-cite-title" id="page-title">Unilateral Calcification of the Caudate and Putamen: Association with Underlying Developmental Venous Anomaly</h1>  
    	<div class="highwire-cite-authors"><span  class="highwire-citation-authors"><span class="highwire-citation-author first" data-delta="0">S. Dehkharghani</span>, <span class="highwire-citation-author" data-delta="1">W.P. Dillon</span>, <span class="highwire-citation-author" data-delta="2">S.O. Bryant</span> and <span class="highwire-citation-author" data-delta="3">N.J. Fischbein</span></span></div>
  
    	<div class="highwire-cite-metadata"><span  class="highwire-cite-metadata-journal highwire-cite-metadata">American Journal of Neuroradiology </span><span  class="highwire-cite-metadata-date highwire-cite-metadata">November 2010,  </span><span  class="highwire-cite-metadata-volume highwire-cite-metadata">31 </span><span  class="highwire-cite-metadata-issue highwire-cite-metadata">(10) </span><span  class="highwire-cite-metadata-pages highwire-cite-metadata">1848-1852; </span><span  class="highwire-cite-metadata-doi highwire-cite-metadata">DOI: https://doi.org/10.3174/ajnr.A2199 </span></div>
  
  
  
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<p id="p-1"><strong>SUMMARY:</strong> Stenosis of a DVA may result in chronic venous ischemia. We present 6 patients (3 men, 3 women; age range, 30–79 years; mean age, 53 years) with unilateral calcification of the caudate and putamen on noncontrast CT. This calcification typically spared the anterior limb of the internal capsule. No patient presented with symptoms referable to the basal ganglia or had an underlying metabolic disorder or other process associated with calcium deposition. All patients subsequently underwent gadolinium-enhanced MR imaging and/or CTA or conventional angiography demonstrating the presence of an adjacent DVA. We hypothesize that chronic venous ischemia in the drainage territory of the DVA causes the abnormal mineralization. Greater recognition of this entity will prevent misinterpretation of this finding as acute hemorrhage and will prevent unnecessary and sometimes invasive evaluation in such patients. Furthermore, this entity should be considered in the differential diagnosis of unilateral basal ganglia hyperattenuation.</p>
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</div><div class="section glossary" id="glossary-1"><h2>Abbreviations</h2><dl id="def-list-1"><dt id="def-item-1">AVM<span class="def-item-dt-sep"></span></dt><dd>arteriovenous malformation<span class="def-item-dd-sep"></span></dd><dt id="def-item-2">C<span class="def-item-dt-sep"></span></dt><dd>caudate<span class="def-item-dd-sep"></span></dd><dt id="def-item-3">Ca<sup>2+</sup><span class="def-item-dt-sep"></span></dt><dd>calcification<span class="def-item-dd-sep"></span></dd><dt id="def-item-4">CTA<span class="def-item-dt-sep"></span></dt><dd>CT angiography<span class="def-item-dd-sep"></span></dd><dt id="def-item-5">DVA<span class="def-item-dt-sep"></span></dt><dd>developmental venous anomaly<span class="def-item-dd-sep"></span></dd><dt id="def-item-6">HA<span class="def-item-dt-sep"></span></dt><dd>headache<span class="def-item-dd-sep"></span></dd><dt id="def-item-7">ICH<span class="def-item-dt-sep"></span></dt><dd>intracerebral hemorrhage<span class="def-item-dd-sep"></span></dd><dt id="def-item-8">L<span class="def-item-dt-sep"></span></dt><dd>left<span class="def-item-dd-sep"></span></dd><dt id="def-item-9">NA<span class="def-item-dt-sep"></span></dt><dd>not applicable<span class="def-item-dd-sep"></span></dd><dt id="def-item-10">P<span class="def-item-dt-sep"></span></dt><dd>putamen<span class="def-item-dd-sep"></span></dd><dt id="def-item-11">R<span class="def-item-dt-sep"></span></dt><dd>right<span class="def-item-dd-sep"></span></dd><dt id="def-item-12">SZ<span class="def-item-dt-sep"></span></dt><dd>seizure</dd></dl></div><ul class="copyright-statement"><li class="fn" id="copyright-statement-1">Copyright © American Society of Neuroradiology</li></ul><span class="highwire-journal-article-marker-end"></span></div><span class="related-urls"></span></div><a href="http://ajnr-stage2.stage.highwire.org/content/31/10/1848.full" class="hw-link hw-link-article-full-text" data-icon-position="" data-hide-link-title="0">View Full Text</a></div>  </div>

  
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      <div class="highwire-cite-title">Unilateral Calcification of the Caudate and Putamen: Association with Underlying Developmental Venous Anomaly</div>  
    	<div class="highwire-cite-authors"><span  class="highwire-citation-authors"><span class="highwire-citation-author first" data-delta="0"><span class="nlm-given-names">S.</span> <span class="nlm-surname">Dehkharghani</span></span>, <span class="highwire-citation-author" data-delta="1"><span class="nlm-given-names">W.P.</span> <span class="nlm-surname">Dillon</span></span>, <span class="highwire-citation-author" data-delta="2"><span class="nlm-given-names">S.O.</span> <span class="nlm-surname">Bryant</span></span>, <span class="highwire-citation-author" data-delta="3"><span class="nlm-given-names">N.J.</span> <span class="nlm-surname">Fischbein</span></span></span></div>
  
    	<div class="highwire-cite-metadata"><span  class="highwire-cite-metadata-journal highwire-cite-metadata">American Journal of Neuroradiology </span><span  class="highwire-cite-metadata-date highwire-cite-metadata">Nov 2010, </span><span  class="highwire-cite-metadata-volume highwire-cite-metadata">31 </span><span  class="highwire-cite-metadata-issue highwire-cite-metadata">(10) </span><span  class="highwire-cite-metadata-pages highwire-cite-metadata">1848-1852; </span><span  class="highwire-cite-metadata-doi highwire-cite-metadata"><span class="label">DOI:</span> 10.3174/ajnr.A2199 </span></div>
  
  
  
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