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PT  - JOURNAL ARTICLE
AU  - Bulakbasi, N.
AU  - Kocaoglu, M.
AU  - Tayfun, C.
AU  - Ucoz, T.
TI  - Transient Splenial Lesion of the Corpus Callosum in Clinically Mild Influenza-Associated Encephalitis/Encephalopathy
DP  - 2006 Oct 01
TA  - American Journal of Neuroradiology
PG  - 1983--1986
VI  - 27
IP  - 9
4099  - http://www.ajnr.org/content/27/9/1983.short
4100  - http://www.ajnr.org/content/27/9/1983.full
SO  - Am. J. Neuroradiol.2006 Oct 01; 27
AB  - BACKGROUND: Reversible lesions in the splenium of the corpus callosum (SCC), caused by various agents such as influenza, rotavirus, Escherichia coli, mumps, and adenovirus, were previously defined in a handful of cases. We present 5 cases with transient diffusion restriction of the SCC associated with influenza A virus infection.MATERIALS AND METHODS: Five patients with influenza-associated encephalitis/encephalopathy and sudden-onset neurologic symptoms following a prodromal flulike episode were examined by MR and diffusion-weighted imaging (DWI).RESULTS: Three patients, who had drowsiness and new-onset convulsions, recovered spontaneously without any medication. In the other 2 seizure-free patients, 1 had trigeminal neuralgia and headache and the other had facial numbness and left upper monoparesis. All patients had round well-defined ovoid hyperintense splenial lesions (14.94 ± 1.87 mm) on DWI with a significantly low apparent diffusion coefficient (ADC) of 0.41 ± 0.05 × 10−3 mm2/s compared with 0.84 ± 0.01 × 10−3 mm2/s of normal-appearing white matter. In the patient with a motor deficit, additional lesions were found in the cerebral deep white matter. The high signal intensity of the splenial and deep white matter lesions on DWI completely disappeared on follow-up studies, and ADC values also improved, returning to those of normal-appearing white matter on days 8–11. Clinically, all patients completely recovered on days 4–9.CONCLUSION: A transient lesion of the SCC is a significant but nonspecific finding. It is probably due to edematous and/or inflammatory changes of the SCC. It may be the only detectable change in patients with good prognosis, indicating a clinically mild form of encephalitis/encephalopathy.