1naresh
Array ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) ) 1nareshArray ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) )Table 1:Comparison of angiographic findings and treatment parameters between patients with HHT and patients with idiopathic epistaxis
Normal Angiogram Required Multiple Embolizations HHT 1/12 (8%) 6/12 (50%) Idiopathic 7/10 (70%) 1/10 (10%) P = .0062 P = 0.0743 Previous surgery for epistaxis HHT 8/12 (67%) Idiopathic 4/10 (40%) P = .3913 Mean number of vessels embolized HHT 2.75 Idiopathic 2.27 P = .1975 Bilateral ECA vessel embolization performed HHT 15/16 (94%) Idiopathic 7/11 (64%) P = .1252
Note:—HHT indicates hereditary hemorrhagic telangiectasia; ECA, external carotid artery. P values were calculated using the Fisher exact test for categoric data and t test for continuous data.