1naresh
Array ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) ) 1nareshArray ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) )Comparison between KTS and PWSa
Feature KTS PWS Types of vascular malformations Slow flow; capillary, lymphatic, venous Fast flow; capillary, arterial, venous Color of capillary stain Red to purplish Pink and diffuse; multiple capillary stains if RASA1 AV fistulas Insignificant Significant Marginal venous anomaly Very common Not found Lymphatic vesicles Present Uncommon Limb affected Upper 5%, lower 95% Upper 23%, lower 77% Prognosis Poor quality of life due to venous insufficiency and complications of lymphatic malformation; risk of pulmonary embolism Progressive symptoms with cutaneous ischemia and possible heart failure
a Modified from Reference 5.