1naresh
Array ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) ) 1nareshArray ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) )TABLE 3:TABLE 3: ADC changes of subcortical low-intensity lesions in 55 patients
ADC (10−3 mm2/s) of Subcortical White Matter SCLI (+) (n = 14) SCLI (−) (n = 41) Lesion 0.68 ± 0.10* 0.72 ± 0.05 Contralateral 0.75 ± 0.08 0.73 ± 0.05 Decrease (%)† 9.3 ± 11.4 0.59 ± 7.59 P value <.00001 >.05
Note.—SCLI (+) and SCLI (−) indicate patients with and those without, respectively, subcortical low intensity.
* ADC of subcortical low-intensity lesion was significantly lower than that of contralateral subcortical white matter (Student t test).
† Percentage decrease in signal intensity of the lesion to the corresponding area in the contralateral hemisphere.