1naresh
Array ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) ) 1nareshArray ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) )TABLE 2:Mean signal intensity ratios of the lesions on ADC maps
Group and Lesion Signal Intensity Ratio At 30 min At 2 h 1, triolein (n = 8) Type 1 (n = 7) 0.72 (0.12) 0.67 (0.12) Type 2 (n = 8) 0.97 (0.16) 1.12 (0.28) 2, oleic acid (n = 10) Type 1 (n = 8) 0.61 (0.08) 0.60 (0.11) Type 2 (n = 10) 0.91 (0.18) 0.95 (0.13)
Note.—The data are the mean ± SD. In type 1 lesions, the signal intensity ratios were significantly lower in group 2 compared with those in group 1 at both 30 minutes and 2 hours (P = .027). However, in type 2 lesions, the signal intensity ratios did not significantly differ between the two groups at either time point (P = .144).