1naresh
Array ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) ) 1nareshArray ( [urn:ac.highwire.org:guest:identity] => Array ( [runtime-id] => urn:ac.highwire.org:guest:identity [type] => guest [service-id] => ajnr-ac.highwire.org [access-type] => Controlled [privilege] => Array ( [urn:ac.highwire.org:guest:privilege] => Array ( [runtime-id] => urn:ac.highwire.org:guest:privilege [type] => privilege-set [privilege-set] => GUEST ) ) [credentials] => Array ( [method] => guest ) ) )Table 2:Difference in signal intensity at the origin segment of symptomatic CCAO at different stages
Hyperintensity (n = 0) Isointensity (n = 7) Hypointensity (n = 0) Heterogeneous Intensity (n = 27) P Value <7 days (No.) (%) 0 (0.0) 0 (0.0) 0 (0.0) 5 (14.7) .69 7–30 days (No.) (%) 0 (0.0) 4 (57.1) 0 (0.0) 16 (47.1) 30–90 days (No.) (%) 0 (0.0) 2 (28.6) 0 (0.0) 10 (29.4) ≥90 days (No.) (%) 0 (0.0) 1 (14.3) 0 (0.0) 3 (8.8)