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<meta name="DC.Title" content="MR of optic papilla protrusion in patients with high intracranial pressure." />
<meta name="DC.Date" content="1996-04-01" />
<meta name="DC.Publisher" content="American Journal of Neuroradiology" />
<meta name="DC.Rights" content="Copyright © American Society of Neuroradiology" />
<meta name="DC.AccessRights" content="restricted" />
<meta name="DC.Description" content="PURPOSE To evaluate the signal characteristics of the optic papilla (optic nerve head) on routine cranial MR images in patients with clinical evidence of optic papilla elevation caused by high intracranial pressure, and to compare these findings with findings in healthy adult volunteers.

METHODS We reviewed retropectively the MR imaging examinations of 15 patients who were referred with objectively decreased visual acuity and funduscopic findings of optic papilla elevation. T1-weighted and T2-weighted axial MR images were obtained by using conventional spinecho acquisitions on 1.5-T MR imagers. In addition, the MR imaging studies in 10 healthy adult volunteers without visual impairment were reviewed as controls.

RESULTS In 10 (67%) of the 15 patients, visual elevation of the optic papilla was shown by MR imaging. In all 15 patients, the MR signal intensity of the optic papilla was hypointense relative to the vitreous of the globe on T2-weighted images. In the healthy volunteer group, the optic papillae were all similarly hypointense relative to the vitreous of the globe on T2-weighted images; however, these optic papillae were flat.

CONCLUSION Clinical examination and MR imaging may show elevation of the optic papilla in patients with high intracranial pressure. When chronic, optic papilla elevation has been shown to correlate well with severe loss of vision. Actual edema of the optic papilla seems to play little role in the physical elevation observed clinically in the chronic stages of this pathologic process." />
<meta name="DC.Contributor" content="J R Jinkins" />
<meta name="DC.Contributor" content="S Athale" />
<meta name="DC.Contributor" content="L Xiong" />
<meta name="DC.Contributor" content="W T Yuh" />
<meta name="DC.Contributor" content="M I Rothman" />
<meta name="DC.Contributor" content="P T Nguyen" />
<meta name="article:published_time" content="1996-04-01" />
<meta name="citation_title" content="MR of optic papilla protrusion in patients with high intracranial pressure." />
<meta name="citation_abstract" lang="en" content="&lt;h3&gt;PURPOSE&lt;/h3&gt;
&lt;p&gt;To evaluate the signal characteristics of the optic papilla (optic nerve head) on routine cranial MR images in patients with clinical evidence of optic papilla elevation caused by high intracranial pressure, and to compare these findings with findings in healthy adult volunteers.&lt;/p&gt;&lt;h3&gt;METHODS&lt;/h3&gt;
&lt;p&gt;We reviewed retropectively the MR imaging examinations of 15 patients who were referred with objectively decreased visual acuity and funduscopic findings of optic papilla elevation. T1-weighted and T2-weighted axial MR images were obtained by using conventional spinecho acquisitions on 1.5-T MR imagers. In addition, the MR imaging studies in 10 healthy adult volunteers without visual impairment were reviewed as controls.&lt;/p&gt;&lt;h3&gt;RESULTS&lt;/h3&gt;
&lt;p&gt;In 10 (67%) of the 15 patients, visual elevation of the optic papilla was shown by MR imaging. In all 15 patients, the MR signal intensity of the optic papilla was hypointense relative to the vitreous of the globe on T2-weighted images. In the healthy volunteer group, the optic papillae were all similarly hypointense relative to the vitreous of the globe on T2-weighted images; however, these optic papillae were flat.&lt;/p&gt;&lt;h3&gt;CONCLUSION&lt;/h3&gt;
&lt;p&gt;Clinical examination and MR imaging may show elevation of the optic papilla in patients with high intracranial pressure. When chronic, optic papilla elevation has been shown to correlate well with severe loss of vision. Actual edema of the optic papilla seems to play little role in the physical elevation observed clinically in the chronic stages of this pathologic process.&lt;/p&gt;" />
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<meta name="citation_publication_date" content="1996/04/01" />
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<meta name="citation_author" content="J R Jinkins" />
<meta name="citation_author" content="S Athale" />
<meta name="citation_author" content="L Xiong" />
<meta name="citation_author" content="W T Yuh" />
<meta name="citation_author" content="M I Rothman" />
<meta name="citation_author" content="P T Nguyen" />
<meta name="twitter:title" content="MR of optic papilla protrusion in patients with high intracranial pressure." />
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<meta name="twitter:description" content="PURPOSE To evaluate the signal characteristics of the optic papilla (optic nerve head) on routine cranial MR images in patients with clinical evidence of optic papilla elevation caused by high intracranial pressure, and to compare these findings with findings in healthy adult volunteers.

METHODS We reviewed retropectively the MR imaging examinations of 15 patients who were referred with objectively decreased visual acuity and funduscopic findings of optic papilla elevation. T1-weighted and T2-weighted axial MR images were obtained by using conventional spinecho acquisitions on 1.5-T MR imagers. In addition, the MR imaging studies in 10 healthy adult volunteers without visual impairment were reviewed as controls.

RESULTS In 10 (67%) of the 15 patients, visual elevation of the optic papilla was shown by MR imaging. In all 15 patients, the MR signal intensity of the optic papilla was hypointense relative to the vitreous of the globe on T2-weighted images. In the healthy volunteer group, the optic papillae were all similarly hypointense relative to the vitreous of the globe on T2-weighted images; however, these optic papillae were flat.

CONCLUSION Clinical examination and MR imaging may show elevation of the optic papilla in patients with high intracranial pressure. When chronic, optic papilla elevation has been shown to correlate well with severe loss of vision. Actual edema of the optic papilla seems to play little role in the physical elevation observed clinically in the chronic stages of this pathologic process." />
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<meta name="og-site-name" property="og:site_name" content="American Journal of Neuroradiology" />
<meta name="og-description" property="og:description" content="PURPOSE To evaluate the signal characteristics of the optic papilla (optic nerve head) on routine cranial MR images in patients with clinical evidence of optic papilla elevation caused by high intracranial pressure, and to compare these findings with findings in healthy adult volunteers.

METHODS We reviewed retropectively the MR imaging examinations of 15 patients who were referred with objectively decreased visual acuity and funduscopic findings of optic papilla elevation. T1-weighted and T2-weighted axial MR images were obtained by using conventional spinecho acquisitions on 1.5-T MR imagers. In addition, the MR imaging studies in 10 healthy adult volunteers without visual impairment were reviewed as controls.

RESULTS In 10 (67%) of the 15 patients, visual elevation of the optic papilla was shown by MR imaging. In all 15 patients, the MR signal intensity of the optic papilla was hypointense relative to the vitreous of the globe on T2-weighted images. In the healthy volunteer group, the optic papillae were all similarly hypointense relative to the vitreous of the globe on T2-weighted images; however, these optic papillae were flat.

CONCLUSION Clinical examination and MR imaging may show elevation of the optic papilla in patients with high intracranial pressure. When chronic, optic papilla elevation has been shown to correlate well with severe loss of vision. Actual edema of the optic papilla seems to play little role in the physical elevation observed clinically in the chronic stages of this pathologic process." />
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      <h1 class="highwire-cite-title" id="page-title">MR of optic papilla protrusion in patients with high intracranial pressure.</h1>  
    	<div class="highwire-cite-authors"><span  class="highwire-citation-authors"><span class="highwire-citation-author first" data-delta="0">J R Jinkins</span>, <span class="highwire-citation-author" data-delta="1">S Athale</span>, <span class="highwire-citation-author" data-delta="2">L Xiong</span>, <span class="highwire-citation-author" data-delta="3">W T Yuh</span>, <span class="highwire-citation-author" data-delta="4">M I Rothman</span> and <span class="highwire-citation-author" data-delta="5">P T Nguyen</span></span></div>
  
    	<div class="highwire-cite-metadata"><span  class="highwire-cite-metadata-journal highwire-cite-metadata">American Journal of Neuroradiology </span><span  class="highwire-cite-metadata-date highwire-cite-metadata">April 1996,  </span><span  class="highwire-cite-metadata-volume highwire-cite-metadata">17 </span><span  class="highwire-cite-metadata-issue highwire-cite-metadata">(4) </span><span  class="highwire-cite-metadata-pages highwire-cite-metadata">665-668; </span></div>
  
  
  
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        	<div class="highwire-cite-metadata"><span  class="highwire-cite-metadata-volume highwire-cite-metadata">Vol. 17</span>, <span  class="highwire-cite-metadata-issue highwire-cite-metadata">Issue 4 </span><div  class="highwire-cite-metadata-pubdate highwire-cite-metadata">1 Apr 1996 </div></div>
      
      
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      <div class="highwire-cite-title">MR of optic papilla protrusion in patients with high intracranial pressure.</div>  
    	<div class="highwire-cite-authors"><span  class="highwire-citation-authors"><span class="highwire-citation-author first" data-delta="0"><span class="nlm-given-names">J R</span> <span class="nlm-surname">Jinkins</span></span>, <span class="highwire-citation-author" data-delta="1"><span class="nlm-given-names">S</span> <span class="nlm-surname">Athale</span></span>, <span class="highwire-citation-author" data-delta="2"><span class="nlm-given-names">L</span> <span class="nlm-surname">Xiong</span></span>, <span class="highwire-citation-author" data-delta="3"><span class="nlm-given-names">W T</span> <span class="nlm-surname">Yuh</span></span>, <span class="highwire-citation-author" data-delta="4"><span class="nlm-given-names">M I</span> <span class="nlm-surname">Rothman</span></span>, <span class="highwire-citation-author" data-delta="5"><span class="nlm-given-names">P T</span> <span class="nlm-surname">Nguyen</span></span></span></div>
  
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